Demonstration of how the dual incretin agonist tirzpetide requires the GIPR in human islets for insulin secretion
Various stressors, including pregnancy, surgery and illness induce a reversible acceleration in biological age.
Ketone flux through BDH1 supports metabolic remodeling of skeletal and cardiac muscles in response to intermittent time-restricted feeding
APOL1 G1 and G2 variants cause kidney disease by increasing Na+/K+ transport across the plasma membrane of podocytes
- Demonstration of how the dual incretin agonist tirzpetide requires the GIPR in human islets for insulin secretion
- Various stressors, including pregnancy, surgery and illness induce a reversible acceleration in biological age.
- Ketone flux through BDH1 supports metabolic remodeling of skeletal and cardiac muscles in response to intermittent time-restricted feeding
- APOL1 G1 and G2 variants cause kidney disease by increasing Na+/K+ transport across the plasma membrane of podocytes
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November 12
Prashant Rajbhandari, Ph.D.
4:00 pm to
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November 19
Alex Banks, Ph.D.
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December 10
Intersecting pathways of mitochondrial metabolism and lipid synthesis
Shawn Burgess, Ph.D.
4:00 pm to
5:00 pm