Causal diagram illustrating challenges in discovery of direct causes and Markov boundaries of longevity
JAK inhibitor blocks COVID-19-cytokine-induced JAK-STAT-APOL1 signaling in glomerular cells and podocytopathy in human kidney organoids.
Significant phenotypic associations with COVID 19 risk alleles in UK Biobank
Steroid-sensitive nephrotic syndrome candidate gene CLVS1 regulates podocyte oxidative stress and endocytosis
- Causal diagram illustrating challenges in discovery of direct causes and Markov boundaries of longevity
- JAK inhibitor blocks COVID-19-cytokine-induced JAK-STAT-APOL1 signaling in glomerular cells and podocytopathy in human kidney organoids.
- Significant phenotypic associations with COVID 19 risk alleles in UK Biobank
- Steroid-sensitive nephrotic syndrome candidate gene CLVS1 regulates podocyte oxidative stress and endocytosis
DUKE MOLECULAR PHYSIOLOGY INSTITUTE
Our mission is to produce integrated multi-omics and physiologic profiles of chronic human diseases, and to use such profiles to develop new disease detection strategies, novel therapies, and insights into disease mechanisms.
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DMPI RESEARCH SECTIONS
